rabbit polyclonal anti s100a10 (Proteintech)
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Proteintech
rabbit polyclonal anti s100a10
Rabbit Polyclonal Anti S100a10, supplied by Proteintech, used in various techniques. Bioz Stars score: 95/100, based on 111 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/rabbit+polyclonal+anti+s100a10/S100A10+Antibody/pm40939434-66-72-76
Average 95 stars, based on 111 article reviews
Rabbit Polyclonal Anti S100a10, supplied by Proteintech, used in various techniques. Bioz Stars score: 95/100, based on 111 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/rabbit+polyclonal+anti+s100a10/S100A10+Antibody/pm40939434-66-72-76
Average 95 stars, based on 111 article reviews
rabbit polyclonal anti s100a10 - by Bioz Stars,
2026-09
95/100 stars
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Incubation:Article Title: The effects of A1/A2 astrocytes on oligodendrocyte linage cells against white matter injury under prolonged cerebral hypoperfusion. Article Snippet: Funding information Takeda Science Foundation; Grants-in-Aid from the Foundation of Strategic Research Projects; Ministry of Education, Culture, Sports, Science and Technology, Japan; JSPS KAKENHI, Grant/Award Number: JP16K07067 Abstract As oligodendrocyte precursor cells (OPCs) are vulnerable to ischemia, their differentiation to oligodendrocytes (OLG) is impaired in chronic cerebral hypoperfusion.. Astrocyte–OLG interaction is important for white matter homeostasis.. Recently, reactive astrocytes were separated into two types, A1 (cytotoxic) and A2 (neurotrophic). Article Title: GRP78 alleviates secondary brain injury by regulating phenotype polarization in astrocytes via JAK2-STAT3 pathway after intracerebral hemorrhage. Article Snippet: Background: Astrocytes, exhibiting dual phenotypes (A1 neurotoxic and A2 neuroprotective), play a pivotal role in secondary brain injury (SBI) subsequent to intracerebral hemorrhage (ICH).. However, the underlying mechanisms remain elusive.. This study sought to explore whether Glucose-regulated protein 78 (GRP78), an essential chaperone involved in endoplasmic reticulum stress, alleviates SBI by influencing astrocytic polarization and to elucidate the underlying mechanisms. Marker:Article Title: The effects of A1/A2 astrocytes on oligodendrocyte linage cells against white matter injury under prolonged cerebral hypoperfusion. Article Snippet: Funding information Takeda Science Foundation; Grants-in-Aid from the Foundation of Strategic Research Projects; Ministry of Education, Culture, Sports, Science and Technology, Japan; JSPS KAKENHI, Grant/Award Number: JP16K07067 Abstract As oligodendrocyte precursor cells (OPCs) are vulnerable to ischemia, their differentiation to oligodendrocytes (OLG) is impaired in chronic cerebral hypoperfusion.. Astrocyte–OLG interaction is important for white matter homeostasis.. Recently, reactive astrocytes were separated into two types, A1 (cytotoxic) and A2 (neurotrophic). Article Title: Astrocytic Extracellular Vesicles Regulated by Microglial Inflammatory Responses Improve Stroke Recovery. Article Snippet: .. The primary antibodies used in this study were goat polyclonal anti-GFAP (1:40,000; Abcam), goat polyclonal anti-C3d (a marker for A1 astrocytes) (1:2,000, R&D Systems), In Vitro:Article Title: GRP78 alleviates secondary brain injury by regulating phenotype polarization in astrocytes via JAK2-STAT3 pathway after intracerebral hemorrhage. Article Snippet: Background: Astrocytes, exhibiting dual phenotypes (A1 neurotoxic and A2 neuroprotective), play a pivotal role in secondary brain injury (SBI) subsequent to intracerebral hemorrhage (ICH).. However, the underlying mechanisms remain elusive.. This study sought to explore whether Glucose-regulated protein 78 (GRP78), an essential chaperone involved in endoplasmic reticulum stress, alleviates SBI by influencing astrocytic polarization and to elucidate the underlying mechanisms. In Vivo:Article Title: GRP78 alleviates secondary brain injury by regulating phenotype polarization in astrocytes via JAK2-STAT3 pathway after intracerebral hemorrhage. Article Snippet: Background: Astrocytes, exhibiting dual phenotypes (A1 neurotoxic and A2 neuroprotective), play a pivotal role in secondary brain injury (SBI) subsequent to intracerebral hemorrhage (ICH).. However, the underlying mechanisms remain elusive.. This study sought to explore whether Glucose-regulated protein 78 (GRP78), an essential chaperone involved in endoplasmic reticulum stress, alleviates SBI by influencing astrocytic polarization and to elucidate the underlying mechanisms. |
